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Methods And Compounds For The Treatment Or Prevention Of Severe Influenza - EP3478322

The patent EP3478322 was granted to Hvivo Services on Dec 30, 2020. The application was originally filed on Jun 26, 2017 under application number EP17736709A. The patent is currently recorded with a legal status of "Granted And Under Opposition".

EP3478322

HVIVO SERVICES
Application Number
EP17736709A
Filing Date
Jun 26, 2017
Status
Granted And Under Opposition
Nov 27, 2020
Publication Date
Dec 30, 2020
External Links
Slate, Register, Google Patents

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GILL JENNINGS & EVERYSep 30, 2021CARPMAELS & RANSFORDWITHDRAWN

Patent Citations (3) New

Patent citations refer to prior patents cited during different phases such as opposition or international search.

Citation PhasePublication Number
INTERNATIONAL-SEARCH-REPORTWO2015173788
OPPOSITIONWO0119322
OPPOSITIONWO2015173788

Non-Patent Literature (NPL) Citations (12) New

NPL citations refer to non-patent references such as research papers, articles, or other publications cited during examination or opposition phases.

Citation PhaseReference Text
OPPOSITION- HUI et al., "Induction of Proinflammatory Cytokines in Primary Human Macrophages by Influenza A Virus (H5N1) Is Selectively Regulated by IFN Regulatory Factor 3 and p38 MAPK", The Journal of Immunology, (20090115), pages 1088 - 1098, XP055849642
OPPOSITION- PAQUETTE et al., "Interleukin-6 Is a Potential Biomarker for Severe Pandemic H1N1 Influenza A Infection", PLoS ONE, (20120605), vol. 7, no. 6, XP055849658
OPPOSITION- RAMOS et al., "Modulating the innate immune response to influenza A virus: potential therapeutic use of anti-inflammatory drugs", Frontiers in Immunology, (20150720), vol. 6, XP055849664
OPPOSITION- WU et al., "Innate immune response to H3N2 and H1N1 influenza virus infection in a human lung organ culture model", Virology, (20100120), pages 178 - 188, XP055849645
OPPOSITION- GEILER et al., "Comparison of pro-inflammatory cytokine expression and cellular signal transduction in human macrophages infected with different influenza A viruses", Med Microbiol Immunol, (20100924), vol. 200, pages 53 - 60, XP019873149
OPPOSITION- LIU et al., "Mouse lung slices: An ex vivo model for the evaluation of antiviral and anti-inflammatory agents against influenza viruses", Antiviral Research, (20150526), vol. 120, pages 101 - 111, XP055355559
OPPOSITION- CUENDA et al., "p38 MAP-Kinases pathway regulation, function and role in human diseases", Biochimica et Biophysica Acta, (20070324), XP022181685
OPPOSITION- CHOI et al., "A novel p38 mitogen activated protein kinase (MAPK) specific inhibitor suppresses respiratory syncytial virus and influenza A virus replication by inhibiting virus-induced p38 MAPK activation", Biochemical and Biophysical Research Communications, (20160623), vol. 477, XP029641769
OPPOSITION- GEILER et al., "N-acetyl-L-cysteine (NAC) inhibits virus replication and expression of pro-inflammatory molecules in A549 cells infected with highly pathogenic H5N1 influenza A virus", Biochemical Pharmacology, (20090902), vol. 79, pages 413 - 420, XP026776992
OPPOSITION- BORGELING et al., "Inhibition of p38 Mitogen-activated Protein Kinase Impairs Influenza Virus-induced Primary and Secondary Host Gene Responses and Protects Mice from Lethal H5N1 Infection", The Journal of Biological Chemistry, (20131104), pages 13 - 27, XP055161048
OPPOSITION- LEE et al., "p38 Mitogen-Activated Protein Kinase-Dependent Hyperinduction of Tumor Necrosis Factor Alpha Expression in Response to Avian Influenza Virus H5N1", Journal of Virology, (20050800), vol. 79, no. 16, XP055577642
OPPOSITION- WEI et al., "Roles of p38 MAPK in the regulation of the inflammatory response to swine influenza virus-induced acute lung injury in mice", Acta virologica, (20140000), XP055577637

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